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Key findings are elevated right-sided pressures (CVP 14) and high pulmonary artery pressures (42/28, mean 33) with a relatively normal PCWP (10), reduced cardiac output (3.0 L/min), and increased SVR. This profile aligns with pulmonary hypertension and right ventricular (RV) failure: pressure overload of the RV raises CVP and PAP, while left-sided filling pressure (PCWP) remains normal or low-normal. Cardiogenic shock from LV failure (A) would typically elevate PCWP. Cardiac tamponade (B) causes equalization of diastolic pressures (CVP, PAP diastolic, PCWP) and low CO. Distributive shock (D) features low SVR and often normal/low filling pressures with high or normal CO in early stages. Management focuses on optimizing RV preload (avoid overdistension), reducing RV afterload (oxygenation, treat hypoxia/hypercapnia, consider inhaled pulmonary vasodilators if available), and carefully using vasopressors/inotropes to maintain perfusion.